Droplet impact and spreading on solid substrates are well understood for Newtonian fluids, yet how viscoelasticity alone modifies the maximal spreading remains unclear. To identify the mechanisms governing the spreading dynamics, we conducted impact experiments and measured the maximal spreading diameter to quantify how fluid elasticity modifies the maximal spreading of impacting droplets. Experiments were performed using fluids within a narrow range of viscosity and surface tension, but with varying relaxation times. For a wide range of conditions, viscoelastic droplets follow a similar behavior as Newtonian ones; however, their maximal spreading diameter is significantly reduced compared with the Newtonian behavior when the Deborah number is of order unity. These observations are rationalized by incorporating the viscoelastic effects into a classical energy balance model. The scaling argument obtained from this model explains the reported reduction in maximal spreading and identifies the range of fluid properties for which the strongest viscoelastic effects emerge.
The study of spreading processes often analyzes networks at different resolutions, e.g., at the level of individuals or countries, but it is not always clear how properties at one resolution can carry over to another. Accordingly, in this work we use dissipativity theory from control system analysis to characterize composite spreading networks that are comprised by many interacting subnetworks. We first develop a method to represent spreading networks that have inputs and outputs. Then we define a composition operation for composing multiple spreading networks into a larger composite spreading network. Next, we develop storage and supply rate functions that can be used to demonstrate that spreading dynamics are dissipative. We then derive conditions under which a composite spreading network will converge to a disease-free equilibrium as long as its constituent spreading networks are dissipative with respect to those storage and supply rate functions. To illustrate these results, we use simulations of an influenza outbreak in a primary school, and we show that an outbreak can be prevented by decreasing the average interaction time between any pair of classes to less than 79% of the
Nonstabilizerness, or magic, constitutes a fundamental resource for quantum computation and a crucial ingredient for quantum advantage. Recent progress has substantially advanced the characterization of magic in many-body quantum systems, with stabilizer Rényi entropy (SRE) emerging as a computable and experimentally accessible measure. In this work, we investigate the spreading of SRE in terms of single-qubit reduced density matrices, where an initial product state that contains magic in a local region evolves under brickwork random Clifford circuits. For the case with Haar-random local Clifford gates, we find that the spreading profile exhibits a diffusive structure within a ballistic light cone when viewed through a normalized version of single-qubit SRE, despite the absence of explicit conserved charges. We further examine the robustness of this non-ballistic behavior of the normalized single-qubit SRE spreading by extending the analysis to a restricted Clifford circuit, where we unveil a superdiffusive spreading. Finally, we discuss that a similar non-ballistic spreading within the light cone is found for another indicator of the magic, i.e., the robustness of magic.
Exploring the internal mechanism of information spreading is critical for understanding and controlling the process. Traditional spreading models often assume individuals play the same role in the spreading process. In reality, however, individuals' diverse characteristics contribute differently to the spreading performance, leading to a heterogeneous infection rate across the system. To investigate network spreading dynamics under heterogeneous infection rates, we integrate two individual-level features -- influence (i.e., the ability to influence neighbors) and susceptibility (i.e., the extent to be influenced by neighbors) -- into the independent cascade model. Our findings reveal significant differences in spreading performance under heterogeneous and constant infection rates, with traditional structural centrality metrics proving more effective in the latter scenario. Additionally, we take the constant and heterogeneous infection rates into a state-of-the-art maximization algorithm, the well-known TIM algorithm, and find the seeds selected by heterogeneous infection rates are more dispersed compared to those under constant rates. Lastly, we find that both individuals' influenc
The spreading behaviour of cohesive sand powder is modelled by Discrete Element Method, and the spreadability and the mechanical jamming are focused. The empty patches and total particle volume of the spread layer are examined, followed by the analysis of the geometry force and jamming structure. The results show that several empty patches with different size and shapes could be observed within the spread layer along the spreading direction even when the gap height increases to 3.0D90. Large particles are more difficult to be spread onto the base due to jamming, although their size is smaller than the gap height. Size segregation of particles occurs before particles entering the gap between the blade and base. There are almost no particles on the smooth base when the gap height is small, due to the full-slip flow of particles. The difference of the spread layer and spreadability between the cases with rough and smooth base is reduced by the increase of the gap height. An interesting correlation between jamming effect and local defects (empty spaces) in the powder layer is identified. The resistance to particle rolling is important for the mechanical jamming reported in this work. T
We study epidemic spreading processes in large networks, when the spread is assisted by a small number of external agents: infection sources with bounded spreading power, but whose movement is unrestricted vis-à-vis the underlying network topology. For networks which are `spatially constrained', we show that the spread of infection can be significantly speeded up even by a few such external agents infecting randomly. Moreover, for general networks, we derive upper-bounds on the order of the spreading time achieved by certain simple (random/greedy) external-spreading policies. Conversely, for certain common classes of networks such as line graphs, grids and random geometric graphs, we also derive lower bounds on the order of the spreading time over all (potentially network-state aware and adversarial) external-spreading policies; these adversarial lower bounds match (up to logarithmic factors) the spreading time achieved by an external agent with a random spreading policy. This demonstrates that random, state-oblivious infection-spreading by an external agent is in fact order-wise optimal for spreading in such spatially constrained networks.
Foam is an industrially important form of matter, commonly deployed to clean objects and even our own skin, thanks to its ability to absorb oil and particles into its interior. To clean a large area, a foam is spread over a substrate, but the optimum conditions and mechanism have been unclear. Here, we study how a foam is spread by a rigid plate on a substrate as a function of spreading velocity, gap height, confinement length, amount of foam and wettability of the substrate. Three distinguishable spreading patterns were found: homogeneous spreading, non-spreading, and slender spreading. It is also found that the dynamics and the mechanism of the spreading can be explained by coupling among dewetting, anchoring, shear stress, viscous stress and yield stress. It is a unique feature of foams, which is not observed in simple liquids and then these findings are also critical for understanding the mechanical response of other soft jamming systems such as cells and emulsions.
Problems of consensus in multi-agent systems are often viewed as a series of independent, simultaneous local decisions made between a limited set of options, all aimed at reaching a global agreement. Key challenges in these protocols include estimating the likelihood of various outcomes and finding bounds for how long it may take to achieve consensus, if it occurs at all. To date, little attention has been given to the case where some agents have no initial opinion. In this paper, we introduce a variant of the consensus problem which includes what we call `agnostic' nodes and frame it as a combination of two known and well-studied processes: voter model and rumour spreading. We show (1) a martingale that describes the probability of consensus for a given colour, (2) bounds on the number of steps for the process to end using results from rumour spreading and voter models, (3) closed formulas for the probability of consensus in a few special cases, along with a polynomial-time algorithm for the case where the number of agnostic vertices is at most logarithmic and (4) that the computational complexity of estimating the probability with a Markov chain Monte Carlo process is $O(n^2 \log
With great theoretical and practical significance, identifying the node spreading influence of complex network is one of the most promising domains. So far, various topology-based centrality measures have been proposed to identify the node spreading influence in a network. However, the node spreading influence is a result of the interplay between the network topology structure and spreading dynamics. In this paper, we build up the systematic method by combining the network structure and spreading dynamics to identify the node spreading influence. By combining the adjacent matrix $A$ and spreading parameter $β$, we theoretical give the node spreading influence with the eigenvector of the largest eigenvalue. Comparing with the Susceptible-Infected-Recovered (SIR) model epidemic results for four real networks, our method could identify the node spreading influence more accurately than the ones generated by the degree, K-shell and eigenvector centrality. This work may provide a systematic method for identifying node spreading influence.
Several concepts that model processes of spreading (of information, disease, objects, etc.) in graphs or networks have been studied. In many contexts, we assume that some vertices of a graph $G$ are contaminated initially, before the process starts. By the $q$-forcing rule, a contaminated vertex having at most $q$ uncontaminated neighbors enforces all the neighbors to become contaminated, while by the $p$-percolation rule, an uncontaminated vertex becomes contaminated if at least $p$ of its neighbors are contaminated. In this paper, we consider sets $S$ that are at the same time $q$-forcing sets and $p$-percolating sets, and call them $(p,q)$-spreading sets. Given positive integers $p$ and $q$, the minimum cardinality of a $(p,q)$-spreading set in $G$ is a $(p,q)$-spreading number, $σ_{(p,q)}(G)$, of $G$. While $q$-forcing sets have been studied in a dozen of papers, the decision version of the corresponding graph invariant has not been considered earlier, and we fill the gap by proving its NP-completeness. This, in turn, enables us to prove the NP-completeness of the decision version of the $(p,q)$-spreading number in graphs for an arbitrary choice of $p$ and $q$. Again, for every
Comparing with single networks, the multiplex networks bring two main effects on the spreading process among individuals. First, the pathogen or information can be transmitted to more individuals through different layers at one time, which enlarges the spreading scope. Second, through different layers, an individual can also transmit the pathogen or information to the same individuals more than once at one time, which makes the spreading more effective. To understand the different roles of the spreading scope and effectiveness, we propose an epidemic model on multiplex networks with link overlapping, where the spreading effectiveness of each interaction as well as the variety of channels (spreading scope) can be controlled by the number of overlapping links. We find that for Poisson degree distribution, increasing the epidemic scope (the first effect) is more efficient than enhancing epidemic probability (the second effect) to facilitate the spreading process. However, for power-law degree distribution, the effects of the two factors on the spreading dynamics become complicated. Enhancing epidemic probability makes pathogen or rumor easier to outbreak in a finite system. But after
It is shown that every conditional spreading sequence can be decomposed into two well behaved parts, one being unconditional and the other being convex block homogeneous, i.e. equivalent to its convex block sequences. This decomposition is then used to prove several results concerning the structure of spaces with conditional spreading bases as well as results in the theory of conditional spreading models. Among other things, it is shown that the space $C(ω^ω)$ is universal for all spreading models, i.e., it admits all spreading sequences, both conditional and unconditional, as spreading models. Moreover, every conditional spreading sequence is generated as a spreading model by a sequence in a space that is quasi-reflexive of order one.
Epidemic spreading phenomena are ubiquitous in nature and society. Examples include the spreading of diseases, information, and computer viruses. Epidemics can spread by local spreading, where infected nodes can only infect a limited set of direct target nodes and global spreading, where an infected node can infect every other node. In reality, many epidemics spread using a hybrid mixture of both types of spreading. In this study we develop a theoretical framework for studying hybrid epidemics, and examine the optimum balance between spreading mechanisms in terms of achieving the maximum outbreak size. We show the existence of critically hybrid epidemics where neither spreading mechanism alone can cause a noticeable spread but a combination of the two spreading mechanisms would produce an enormous outbreak. Our results provide new strategies for maximising beneficial epidemics and estimating the worst outcome of damaging hybrid epidemics.
Spreading dynamics of information and diseases are usually analyzed by using a unified framework and analogous models. In this paper, we propose a model to emphasize the essential difference between information spreading and epidemic spreading, where the memory effects, the social reinforcement and the non-redundancy of contacts are taken into account. Under certain conditions, the information spreads faster and broader in regular networks than in random networks, which to some extent supports the recent experimental observation of spreading in online society [D. Centola, Science {\bf 329}, 1194 (2010)]. At the same time, simulation result indicates that the random networks tend to be favorable for effective spreading when the network size increases. This challenges the validity of the above-mentioned experiment for large-scale systems. More significantly, we show that the spreading effectiveness can be sharply enhanced by introducing a little randomness into the regular structure, namely the small-world networks yield the most effective information spreading. Our work provides insights to the understanding of the role of local clustering in information spreading.
The spreading of surfactants on thin films is an industrially and medically important phenomenon, but the dynamics are highly nonlinear and visualization of the surfactant dynamics has been a long-standing experimental challenge. We perform the first quantitative, spatiotemporally-resolved measurements of the spreading of an insoluble surfactant on a thin fluid layer. During the spreading process, we directly observe both the radial height profile of the spreading droplet and the spatial distribution of the fluorescently-tagged surfactant. We find that the leading edge of spreading circular layer of surfactant forms a Marangoni ridge in the underlying fluid, with a trough trailing the ridge as expected. However, several novel features are observed using the fluorescence technique, including a peak in the surfactant concentration which trails the leading edge, and a flat, monolayer-scale spreading film which differs from concentration profiles predicted by current models. Both the Marangoni ridge and surfactant leading edge can be described to spread as $R \propto t^δ$. We find spreading exponents, $δ_H \approx 0.30$ and $δ_Γ\approx 0.22$ for the ridge peak and surfactant leading ed
We examine theoretically the spreading of a viscous liquid drop over a thin film of uniform thickness, assuming the liquid's viscosity is regulated by the concentration of a solute that is carried passively by the spreading flow. The solute is assumed to be initially heterogeneous, having a spatial distribution with prescribed statistical features. To examine how this variability influences the drop's motion, we investigate spreading in a planar geometry using lubrication theory, combining numerical simulations with asymptotic analysis. We assume diffusion is sufficient to suppress solute concentration gradients across but not along the film. The solute field beneath the bulk of the drop is stretched by the spreading flow, such that the initial solute concentration immediately behind the drop's effective contact lines has a long-lived influence on the spreading rate. Over long periods, solute swept up from the precursor film accumulates in a short region behind the contact line, allowing patches of elevated viscosity within the precursor film to hinder spreading. A low-order model provides explicit predictions of the variances in spreading rate and drop location, which are validate
We report experimental results on the rift formation between two freezing wax plates. The plates were pulled apart with constant velocity, while floating on the melt, in a way akin to the tectonic plates of the earth's crust. At slow spreading rates, a rift, initially perpendicular to the spreading direction, was found to be stable, while above a critical spreading rate a "spiky" rift with fracture zones almost parallel to the spreading direction developed. At yet higher spreading rates a second transition from the spiky rift to a zig-zag pattern occurred. In this regime the rift can be characterized by a single angle which was found to be dependent on the spreading rate. We show that the oblique spreading angles agree with a simple geometrical model. The coarsening of the zig-zag pattern over time and the three-dimensional structure of the solidified crust are also discussed.
Promoting information spreading is a booming research topic in network science community. However, the exiting studies about promoting information spreading seldom took into account the human memory, which plays an important role in the spreading dynamics. In this paper we propose a non-Markovian information spreading model on complex networks, in which every informed node contacts a neighbor by using the memory of neighbor's accumulated contact numbers in the past. We systematically study the information spreading dynamics on uncorrelated configuration networks and a group of $22$ real-world networks, and find an effective contact strategy of promoting information spreading, i.e., the informed nodes preferentially contact neighbors with small number of accumulated contacts. According to the effective contact strategy, the high degree nodes are more likely to be chosen as the contacted neighbors in the early stage of the spreading, while in the late stage of the dynamics, the nodes with small degrees are preferentially contacted. We also propose a mean-field theory to describe our model, which qualitatively agrees well with the stochastic simulations on both artificial and real-wor
We present a novel experimental system that can be used to study the dynamics of picoliter droplet spreading over substrates with topographic variations. We concentrate on spreading of a droplet within a recessed stadium-shaped pixel, with applications to the manufacture of POLED displays, and find that the sloping side wall of the pixel can either locally enhance or hinder spreading depending on whether the topography gradient ahead of the contact line is positive or negative. Locally enhanced spreading occurs via the formation of thin pointed rivulets along the side walls of the pixel through a mechanism similar to capillary rise in sharp corners. We demonstrate that a thin-film model combined with an experimentally measured spreading law, which relates the speed of the contact line to the contact angle, provides excellent predictions of the evolving liquid morphologies. We also show that the spreading can be adequately described by a Cox-Voinov law for the majority of the evolution. The model does not include viscous effects and hence, the timescales for the propagation of the thin pointed rivulets are not captured. Nonetheless, this simple model can be used very effectively to
We explain a possible mechanism of an information spreading on a network which spreads extremely far from a seed node, namely the viral spreading. On the basis of a model of the information spreading in an online social network, in which the dynamics is expressed as a random multiplicative process of the spreading rates, we will show that the correlation between the spreading rates enhances the chance of the viral spreading, shifting the tipping point at which the spreading goes viral.